The rebound effect, also known as the Yo-Yo effect, when an individual after diet regains the original weight he had and in some cases up to a certain weight, regardless of the dietary intervention the driver has used to lose weight.
Weight, nutrition and metabolism are regulated by various hormones that act in adipose tissue, the brain and other organs, so it is thought that this weight gain is not only associated with the person who did it. To changes in their eating habits, the type of Diet is not bad, but it could also be associated with changes, both metabolic and physiological, in the body to compensate for this period of “hamburger” by the body, and interprets weight loss as “threat”.
- To avoid an effective rebound it is important that the diet is always accompanied by a doctor or nutritionist.
- So that it adapts to the needs of each person.
- In addition to being able to track their progress.
- That’s also important:.
Similarly, it is important to avoid physical inactivity and maintain physical activity at least 3 times a week for 1 hour.
There are several theories in this regard and it is thought that several factors may be associated:
It is believed that achieving highly restrictive, monotonous and nutritionally unbalanced diets could promote effective rebound in a wide range.
In the case of restrictive diets, it is possible that when starting a normal diet, a nutritious response is created, in which the body seeks to recover the lost, a response to the “burger” that is during this period, could cause metabolic changes that increase the production and accumulation of fats, decrease blood sugar and therefore increase appetite and food intake.
Carbohydrates, proteins and fats during metabolism stimulate oxygen consumption in a different way, so in the case of unbalanced diets where more nutrients predominate than others, such as in the ketogenic diet, for example, you may have something that influences weight gain.
Adipose tissue cells are evacuated when an individual weighs, however, their size and number of cells are maintained for an extended period of time. This is another theory, where adipose tissue cells are thought to remain there for a while, activating compensation mechanisms in the body that gradually grow these cells to a smaller size than their “ordinary” size.
There are several hormones involved in the satiety process, they have been found in people with severe weight loss as levels of leptin, YY peptide, cholecystokinin and insulin are reduced, inhabiting an increase in levels of greline and pancreatic polypeptide.
It is believed that all these hormonal changes help to regain weight (except for an increase in pancreatic polypeptide), which produces an increase in appetite, favoring food intake and therefore weight gain.
To understand what is happening, it is important to explain that greline is a hormone that stimulates appetite in the brain, so its levels are high during fasting periods. Unlike greline, leptin is responsible for reducing appetite, and in people who have lost a considerable weight of more than 5% of their weight, levels of this hormone in the blood are reduced. This situation activates compensation mechanisms and reduces energy expenditure and promotes weight recovery.
In addition to the change in satiety hormones, weight loss has also been associated with changes in the hypothalamus and pituitary gland that can promote an effective rebound.
Some people refer to an increase in appetite after weight loss, which could be associated with any physiological changes that occur in the body; however, if one believes there is more, and that people also understand the gratification of eating an increase in food, encouraging greater consumption.
Some studies have shown that approximately 30 to 35% of weight loss recovers after treatment and 50% of people will regain their initial weight within the fifth year after weight loss.